AnthonyTruth144K shares information related to two teenage boys dying in their sleep after the 2nd dose of the Abomination Shots revealing features resembling a catecholamine-induced injury, not typical myocarditis pathology.
Context.– Myocarditis in adolescents has been diagnosed clinically following the administration of the second dose of an mRNA vaccine for coronavirus disease 2019 (COVID-19).
Objective.– To examine the autopsy microscopic cardiac findings in adolescent deaths that occurred shortly following administration of the second Pfizer-BioNTech COVID-19 dose to determine if the “myocarditis” described in these instances has the typical histopathology of myocarditis.
Design.– Clinical and autopsy investigation of two teenage boys who died shortly following administration of the second Pfizer-BioNTech COVID-19 dose.
Results.– The microscopic examination revealed features resembling a catecholamine-induced injury, not typical myocarditis pathology.
Conclusions.– The myocardial injury seen in these post-vaccine hearts is different from typical myocarditis and has an appearance most closely resembling a catecholamine-mediated stress (toxic) cardiomyopathy. Understanding that these instances are different from typical myocarditis and that cytokine storm has a known feedback loop with catecholamines may help guide screening and therapy.
Catecholamines are hormones made by your adrenal glands, two small glands located above your kidneys. These hormones are released into the body in response to physical or emotional stress. The main types of catecholamines are dopamine, norepinephrine, and epinephrine. Epinephrine is also known as adrenaline.
Catecholamines have an essential role in the physiological regulation of the cardiovascular, respiratory, metabolic, and immune systems—those most heavily affected by COVID-19. Phaeochromocytoma and paraganglioma (PPGL) are chromaffin cell tumours that produce and secrete catecholamines, namely adrenaline and noradrenaline, often in supraphysiological amounts.6 Consequently, catecholamine excess states such as PPGL can cause substantial dysregulation of physiological systems, and lead to pronounced changes in pulmonary (vasoplegia), coronary (myocardial infarction), cerebrovascular (stroke), and remaining systemic vascular tone (hypertension), as well as myocardial disease (cardiomyopathies), tachyarrhythmias (benign and fatal), hypercoagulability (thromboembolism), immune dysregulation (cytokine storm), and diabetogenic states; these outcomes are the same as the risk factors that lead to adverse outcomes from COVID-19.2, 6, 7, 8, 9, 10, 11, 12 These findings suggest that catecholamines might be key mediators in COVID-19. As PPGL is an extensively studied catecholamine excess state, we believe that it might serve as a practical model for exploring the pathological and beneficial actions of catecholamines in patients with COVID-19.
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